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Molecular mechanisms linking wound inflammation and fibrosis: knockdown of osteopontin leads to rapid repair and reduced scarring

Mori, R; Shaw, TJ; Martin, P (2008) Molecular mechanisms linking wound inflammation and fibrosis: knockdown of osteopontin leads to rapid repair and reduced scarring. JOURNAL OF EXPERIMENTAL MEDICINE, 205 (1). 43 - 51 (9). ISSN 0022-1007 https://doi.org/10.1084/jem.20071412
SGUL Authors: Shaw, Tanya J

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Item Type: Article
Additional Information: PubMed ID: 18180311
Keywords: 3T3 Cells, Animals, Cicatrix, Extracellular Matrix, Fibroblasts, Fibrosis, Inflammation, Integrin-Binding Sialoprotein, Leukocytes, Macrophages, Mast Cells, Mice, Models, Biological, Neutrophils, Oligonucleotides, Antisense, Osteopontin, Rats, Sialoglycoproteins, Wound Healing, Science & Technology, Life Sciences & Biomedicine, Immunology, Medicine, Research & Experimental, Research & Experimental Medicine, GROWTH-FACTORS, PULMONARY-FIBROSIS, MAST-CELLS, IN-VIVO, MICE, INFILTRATION, RECRUITMENT, EXPRESSION, IMATINIB, ALPHA
Journal or Publication Title: JOURNAL OF EXPERIMENTAL MEDICINE
ISSN: 0022-1007
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Dates:
DateEvent
21 January 2008Published
Web of Science ID: WOS:000252507100007
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URI: https://openaccess.sgul.ac.uk/id/eprint/2479
Publisher's version: https://doi.org/10.1084/jem.20071412

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