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Sustained activation of XBP1 splicing leads to endothelial apoptosis and atherosclerosis development in response to disturbed flow

Zeng, L; Zampetaki, A; Margariti, A; Pepe, AE; Alam, S; Martin, D; Xiao, Q; Wang, W; Jin, ZG; Cockerill, G; et al. Zeng, L; Zampetaki, A; Margariti, A; Pepe, AE; Alam, S; Martin, D; Xiao, Q; Wang, W; Jin, ZG; Cockerill, G; Mori, K; Li, YS; Hu, Y; Chien, S; Xu, Q (2009) Sustained activation of XBP1 splicing leads to endothelial apoptosis and atherosclerosis development in response to disturbed flow. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 106 (20). 8326 - 8331 (6). ISSN 0027-8424 https://doi.org/10.1073/pnas.0903197106
SGUL Authors: Cockerill, Gillian Wyndham

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Item Type: Article
Additional Information: PubMed ID: 19416856
Keywords: Animals, Apolipoproteins E, Apoptosis, Arteries, Atherosclerosis, Cells, Cultured, DNA-Binding Proteins, Endothelial Cells, Endothelium, Vascular, Humans, Mice, Mice, Knockout, Protein Splicing, Regional Blood Flow, Transcription Factors, Science & Technology, Multidisciplinary Sciences, Science & Technology - Other Topics, caspase, endothelial integrity, Ve-cadherin, vessel graft, mouse model, UNFOLDED PROTEIN RESPONSE, LAMINAR SHEAR-STRESS, TRANSCRIPTION FACTOR XBP-1, NITRIC-OXIDE SYNTHASE, SMOOTH-MUSCLE-CELLS, ENDOPLASMIC-RETICULUM, MESSENGER-RNA, ER STRESS, PROGENITOR CELLS, VE-CADHERIN, caspase, endothelial integrity, Ve-cadherin, vessel graft, mouse model
SGUL Research Institute / Research Centre: Academic Structure > Molecular and Clinical Sciences Research Institute (MCS)
Academic Structure > Molecular and Clinical Sciences Research Institute (MCS) > Vascular (INCCVA)
Journal or Publication Title: PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
ISSN: 0027-8424
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Dates:
DateEvent
19 May 2009Published
Web of Science ID: WOS:000266209000044
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URI: http://openaccess.sgul.ac.uk/id/eprint/2520
Publisher's version: https://doi.org/10.1073/pnas.0903197106

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