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ISG56/IFIT1 is primarily responsible for interferon-induced changes to patterns of parainfluenza virus type 5 transcription and protein synthesis

Andrejeva, J; Norsted, H; Habjan, M; Thiel, V; Goodbourn, S; Randall, RE (2013) ISG56/IFIT1 is primarily responsible for interferon-induced changes to patterns of parainfluenza virus type 5 transcription and protein synthesis. JOURNAL OF GENERAL VIROLOGY, 94. 59 - 68 (10). ISSN 0022-1317 https://doi.org/10.1099/vir.0.046797-0
SGUL Authors: Goodbourn, Stephen Edward

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Item Type: Article
Additional Information: PubMed ID: 23052390
Keywords: Animals, Carrier Proteins, Cell Line, Cell Line, Tumor, Cercopithecus aethiops, DNA Replication, Gene Knockdown Techniques, Humans, Interferon-alpha, Protein Biosynthesis, RNA, Messenger, RNA, Viral, Recombinant Proteins, Respirovirus, Respirovirus Infections, Transcription, Genetic, Vero Cells, Viral Proteins, Virus Replication, Science & Technology, Life Sciences & Biomedicine, Biotechnology & Applied Microbiology, Virology, SIMIAN-VIRUS-5 V-PROTEIN, INDUCED ANTIVIRAL STATE, IFN-BETA PROMOTER, INCLUSION-BODIES, RNA HELICASE, INDUCTION, PARAMYXOVIRUSES, DEGRADATION, ACTIVATION, INHIBIT
SGUL Research Institute / Research Centre: Academic Structure > Infection and Immunity Research Institute (INII)
Journal or Publication Title: JOURNAL OF GENERAL VIROLOGY
ISSN: 0022-1317
Dates:
DateEvent
1 January 2013Published
Web of Science ID: WOS:000315608200006
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URI: https://openaccess.sgul.ac.uk/id/eprint/101282
Publisher's version: https://doi.org/10.1099/vir.0.046797-0

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